Nature Neuroscience, Published online: 21 October 2025; doi:10.1038/s41593-025-02049-3Zeng et al. show that TDP-43, known for repressing cryptic exon usage in frontotemporal dementia/amyotrophic lateral sclerosis, also controls alternative polyadenylation, impacting expression of disease-linked genes (SFPQ, NEFL and TMEM106B).
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This post is Copyright: | October 21, 2025
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