Nature Neuroscience, Published online: 11 March 2025; doi:10.1038/s41593-025-01896-4Cheng et al. identify a mitochondrial complex IV (CIV) deficiency in the brains of patients with sporadic amyotrophic lateral sclerosis (ALS). They demonstrate that defects in mitochondrial CIV induce ALS-like phenotypes in rats and highlight CIV deficiency as a potential risk factor and therapeutic target for ALS.
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This post is Copyright: Man Cheng | March 11, 2025
Nature Neuroscience