Nature Neuroscience, Published online: 10 September 2024; doi:10.1038/s41593-024-01746-9Defective neoangiogenesis and blood–brain barrier leakiness are pathological hallmarks of neuroinflammation. Here the authors show that vascular endothelial growth factor A (VEGF-A) promotes venous endothelial cell proliferation, resulting in the formation of leaky vessels around demyelinating lesions in multiple sclerosis and experimental autoimmune encephalomyelitis.
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This post is Copyright: Sanjid Shahriar | September 10, 2024
Nature Neuroscience