by Clinical Neuropsychologist Online | Monday, August 10, 2026 | Dementia
Abstract INTRODUCTION Subjective cognitive concerns frequently diverge from objective cognitive performance in cognitively unimpaired (CU) older adults, yet the neurobiological basis of this mismatch remains unclear. METHODS In 648 participants from the Investigating...
by Clinical Neuropsychologist Online | Saturday, August 8, 2026 | Dementia
Alzheimer’s &Dementia, Volume 22, Issue 8, August 2026. If you do not see content above, kindly GO TO SOURCE. Not all publishers encode content in a way that enables republishing at Neuro.vip. This post is Copyright: | August 8, 2026 Neuro-Dementia Previous...
by Clinical Neuropsychologist Online | Saturday, August 8, 2026 | Dementia
Abstract INTRODUCTION An extra copy of the amyloid precursor protein (APP) gene causes autosomal dominant Alzheimer’s disease (AD) and AD in Down syndrome (DS), but the factors underlying variability in age at onset (AAO) remain unclear. We investigated whether...
by Clinical Neuropsychologist Online | Saturday, August 8, 2026 | Dementia
Abstract INTRODUCTION Dysfunctional microRNAs and GABAergic interneurons are features of Alzheimer’s disease (AD). The role of neuronal microRNA155 (miR155), elevated in both AD and Down syndrome (DS), remains unknown. METHODS We utilized in silico analyses of...
by Clinical Neuropsychologist Online | Saturday, August 8, 2026 | Dementia
Abstract Over the past decade, the Model Organism Development and Evaluation for Late-Onset Alzheimer’s Disease (MODEL-AD) consortium has transformed preclinical Alzheimer’s disease (AD) research by addressing critical limitations in traditional mouse...
by Clinical Neuropsychologist Online | Friday, August 7, 2026 | Dementia
Abstract INTRODUCTION Limbic-predominant age-related transactive response DNA-binding protein 43 (TDP-43) encephalopathy neuropathological change (LATE-NC) is a cause of dementia resembling Alzheimer’s disease (AD). The 90+ Study found women using hormone...
