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Unlocking Blood Drivers of Neurodegeneration: Mechanisms and therapies

by Clinical Neuropsychologist | Friday, January 3, 2025 | Dementia

Abstract Background Cerebrovascular alterations and innate immune activation are key features of Alzheimer’s disease (AD). However, the mechanisms that link blood-brain barrier disruption to neurodegeneration are poorly understood and well-defined druggable targets at...

Circulating microRNAs related to cognitive performance in the general population

by Clinical Neuropsychologist | Friday, January 3, 2025 | Dementia

Abstract Background MicroRNAs have been linked to dementia. However, understanding their relation to cognition in the general population is required to determine their potential use for the detection and prevention of age-associated cognitive decline and preclinical...

Whole genome methylation sequencing in blood from persons with mild cognitive impairment and dementia due to Alzheimer’s disease identifies cognitive status

by Clinical Neuropsychologist | Thursday, January 2, 2025 | Dementia

Abstract INTRODUCTION Whole genome methylation sequencing (WGMS) in blood identifies differential DNA methylation in persons with late-onset dementia due to Alzheimer’s disease (AD) but has not been tested in persons with mild cognitive impairment (MCI). METHODS...

Withdrawn: Understanding the progression of Alzheimer’s Disease in 5XFAD transgenic female mice using 1H‐[13C]‐NMR Spectroscopy

by Clinical Neuropsychologist | Wednesday, January 1, 2025 | Dementia

Alzheimer’s &Dementia, EarlyView. If you do not see content above, kindly GO TO SOURCE. Not all publishers encode content in a way that enables republishing at Neuro.vip. This post is Copyright: | January 1, 2025 Wiley: Alzheimer’s & Dementia:...

Spatial proteomic differences in chronic traumatic encephalopathy, Alzheimer’s disease, and primary age‐related tauopathy hippocampi

by Clinical Neuropsychologist | Tuesday, December 31, 2024 | Dementia

Abstract INTRODUCTION Alzheimer’s disease (AD), primary age-related tauopathy (PART), and chronic traumatic encephalopathy (CTE) all feature hyperphosphorylated tau (p-tau)–immunoreactive neurofibrillary degeneration, but differ in neuroanatomical distribution...
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